Free Autonomic Pharmacology MCQs with Answers

• A. Morphine
• B. Propanolol
• C. Stropine
• D. Nitroglycerine

Explanation: Nitroglycerin is not contraindicated in bronchial asthma and may relieve associated cardiac ischaemia.

• A. Amphetamine
• B. Nor epinephrine
• C. Isoproterenol
• D. Phenylephrine

Explanation: Isoproterenol stimulates β2 receptors strongly and directly relaxes bronchial smooth muscle, producing marked bronchodilation.

• A. Renal colic
• B. Organophosphorus poisoning
• C. Bronchial asthma
• D. miosisConsult Online Doctors

Explanation: Ipratropium blocks muscarinic receptors in the airways, producing bronchodilation and helping control bronchospasm in asthma and COPD.

• A. Albuterol
• B. Salmeterol
• C. Pirlbuterol
• D. Orciprenaline

Explanation: Salmeterol is a long-acting selective beta2 agonist used for maintenance bronchodilation.

• A. Adrenaline
• B. propranolol
• C. Atropine
• D. Salbutamol

Explanation: Propranolol blocks beta2 receptors in bronchial smooth muscle, removing bronchodilator sympathetic influence and potentially producing…

• A. Histamine
• B. Salbutamol
• C. Acetylcholine
• D. Serotonin

Explanation: Salbutamol is a selective beta2 agonist that relaxes bronchial smooth muscle and relieves bronchospasm.

• A. Bronchial asthma
• B. cardiac asthma
• C. Hypertension
• D. Angina pectrosis

Explanation: Beta2 stimulation relaxes bronchial smooth muscle, so beta2 agonists such as salbutamol are used for bronchial asthma.

• A. Epinephrine
• B. Atropine
• C. A diuretic
• D. A potent vasadilatior

Explanation: Atropine is the usual first-line drug for symptomatic bradycardia because it blocks vagal muscarinic effects on the sinoatrial and…

• A. Penetrate the blood brain barrier
• B. Cause euphoria
• C. Both of the above
• D. None of the above

Explanation: Some indirect sympathomimetics, especially amphetamine, cross the blood-brain barrier and increase central catecholamines, producing…

• A. Blocking acetylcholine receptors
• B. Prevent release of acetyl choline
• C. Destroys acetyl choline
• D. Inactivate acetylcholine esterase enzyme

Explanation: d-Tubocurarine is a competitive, non-depolarising neuromuscular blocker that occupies nicotinic acetylcholine receptors at the motor end…

• A. d-tubecurarine
• B. Acetycholine
• C. Atracurium
• D. Acetazolamide

Explanation: Atracurium undergoes organ-independent Hofmann elimination at physiological temperature and pH.

• A. Atropine
• B. Neostigmine
• C. Scopalamine
• D. Ephedrine

Explanation: Neostigmine inhibits acetylcholinesterase, increasing acetylcholine at muscarinic sites and thereby stimulating salivary secretion.

• A. Organophosphate poisoning
• B. Chronic glaucoma
• C. Syncope
• D. Trigeminal neuralgia

Explanation: Atropine is a muscarinic antagonist that counteracts the excessive acetylcholine effects of organophosphates, especially bronchial…

• A. Epinephrine
• B. Succinylcholine
• C. Tetracycline
• D. Sulfonamides

Explanation: Epinephrine is used therapeutically in severe allergic reactions and anaphylaxis because it rapidly produces bronchodilation…

• A. Anticholinesterases
• B. Alpha-adrenergic blocking agents
• C. Indirect acting sympathomimetics
• D. Direct acting sympathomimetics

Explanation: Ephedrine, tyramine, and amphetamine increase adrenergic activity mainly by releasing stored noradrenaline, so they are indirect-acting…

• A. Motion sickness
• B. Hypertension
• C. Decreased secretions
• D. A & C

Explanation: Scopolamine is an antimuscarinic drug that prevents motion sickness and decreases glandular secretions.

• A. Succinylcholine
• B. Pancuronium
• C. Mivacurum
• D. Decamethonium

Explanation: Mivacurium is a short-acting, non-depolarising neuromuscular blocker, rapidly hydrolysed by plasma cholinesterase.

• A. Chlorozoxazone
• B. Methocarbomol
• C. Succinyl choline
• D. Cortisopradol

Explanation: Succinylcholine acts peripherally at nicotinic neuromuscular junctions and produces skeletal muscle paralysis.

• A. Nicotinic
• B. Muscarinic
• C. Alpha 1 and 2
• D. Beta 1 and 2

Explanation: Muscarinic receptors are activated by acetylcholine and blocked by atropine.

• A. Increases salivary secretions
• B. Decrease salivary secreations
• C. Indicator dye for sialography
• D. Medication for over function

Explanation: Sialogogues stimulate salivary secretion, either by directly activating cholinergic pathways or by promoting parasympathetic activity.